Chiro.Org is proud to have supported CMCC and the AECC for their continuous research
into the health benefits of chiropractic care. Please offer them your financial support.
Chiropractic Research Results for Parkinson's Disease
Chiropractic Management of an 81-Year-Old Man With
Parkinson Disease Signs and Symptoms
Journal of Chiropractic Medicine 2014 (Jun); 13 (2): 116–120 ~ FULL TEXT
This case report describes the responses of a patient with PD who was treated with blue-lensed glasses, vibration stimulation therapy, spinal manipulation, and eye-movement exercises. No definitive conclusions can be drawn from this case; however, it does suggest that the use of chiropractic care may benefit a patient with PD.
Reduction in Symptoms Related to Parkinson's Disease Concomitant with
Subluxation Reduction Following Upper Cervical Chiropractic Care
Journal of Upper Cervical Chiropractic Research 2011 (Mar 14); 18–21
A 67 year-old female patient presenting to a private practice with an atlas subluxation complex as well as signs and symptoms of Parkinson’s disease that include weakness, tremors, scoliosis and rigidity. Over a period of 6 months, the patient was seen 19 times and was adjusted 12 times following the NUCCA protocol. Improvements in radiographic measurements, paraspinal thermography, and sEMG were recorded. Patient self-reported improvements in weakness, tremors, rigidity, and overall mobility. Conclusion: The upper cervical subluxation may be a contributing factor to the symptomatic expression of Parkinson’s disease. Reduction of the subluxation with specific vectored correction may be a plausible, safe, and effective approach for managing PD. More research is warranted investigating the effects of upper cervical care and Parkinson’s Disease.
Upper Cervical Chiropractic Management of 10 Parkinson's Disease Patients
Todays Chiropractic 2000; 29 (4) July ~ FULL TEXT
After a minimum of three months of upper cervical chiropractic care, eight out of 10 cases reviewed showed improvements in Parkinson’s disease and associated symptoms. To my knowledge, these are the first cases reported on this topic since B.J. Palmer’s research 70 years ago. Further investigation into upper cervical injury and resulting neuropatho-physiology as a possible etiology or contributing factor to Parkinson’s disease should be pursued.
Other Management Approaches for Parkinson's Disease
Coffee Deters Parkinson's Disease
Webster Ross, M.D., of the University of Hawaii School of Medicine, explored the connection between caffeine and Parkinson's disease using data from the prospective longitudinal Honolulu Heart Program. In this study, Ross followed 8,004 men of Japanese ancestry living in Honolulu from 1968 to 1996. At the beginning of the study, the average age was 53 years. Consumption of coffee and noncoffee caffeine sources were assessed at the beginning of the study and again in the early 1970s. During the 30 years of follow-up, 102 men developed Parkinson's disease at an average age of 74 years. Coffee drinkers were much less apt to develop the condition, and the more cups of coffee they drank a day, the lower their risk fell. Noncoffee drinkers' risk of Parkinson's disease was more than five times that of men drinking 28 ounces or more of coffee daily. This finding is independent of other dietary factors such as smoking, consumption of milk and sugar, alcohol, and nutrients in coffee besides caffeine. Noncoffee caffeine consumption also lowered Parkinson's disease risk.
Parkinson's Disease as Multifactorial Oxidative Neurodegeneration:
Implications for Integrative Management
Alternative Medicine Review 2000 (Dec); 5 (6): 502–545 ~ FULL TEXT
Parkinson's disease (PD) is the most common movement pathology, severely afflicting dopaminergic neurons within the substantia nigra (SN) along with non-dopaminergic, extra-nigral projection bundles that control circuits for sensory, associative, premotor, and motor pathways. Clinical, experimental, microanatomic, and biochemical evidence suggests PD involves multifactorial, oxidative neurodegeneration, and that levodopa therapy adds to the oxidative burden. The SN is uniquely vulnerable to oxidative damage, having a high content of oxidizable dopamine, neuromelanin, polyunsaturated fatty acids, and iron, and relatively low antioxidant complement with high metabolic rate. Oxidative phosphorylation abnormalities impair energetics in the SN mitochondria, also intensifying oxygen free radical generation. These pro-oxidative factors combine within the SN dopaminergic neurons to create extreme vulnerability to oxidative challenge. Epidemiologic studies and long-term tracking of victims of MPTP (1-methyl-4-phenyl-1,2,3,6,-tetrahydropyridine) poisoning, suggest oxidative stress compounded by exogenous toxins may trigger the neurodegenerative progression of PD. Rational, integrative management of PD requires: (1) dietary revision, especially to lower calories; (2) rebalancing of essential fatty acid intake away from pro-inflammatory and toward anti-inflammatory prostaglandins; (3) aggressive repletion of glutathione and other nutrient antioxidants and cofactors; (4) energy nutrients acetyl L-carnitine, coenzyme Q10, NADH, and the membrane phospholipid phosphatidylserine (PS); (5) chelation as necessary for heavy metals; and (6) liver P450 detoxification support.